Keywords:
Kidney, Abdomen, Ultrasound, Education, Experimental investigations, Normal variants, Congenital, Epidemiology
Authors:
A. D. Tarnoki1, D. L. Tarnoki1, P. Bata1, L. Littvay1, Z. Garami2, K. Karlinger1, V. Bérczi1; 1Budapest/HU, 2Houston, TX/US
DOI:
10.1594/ecr2013/C-1529
Purpose
The decrease in renal cortical thickness (RCT) is an important sign of renal disease.
Several renal conditions (eg.
chronic kidney disease,
renal transplantation,
nephropathia epidemica) are associated with decrease in RCT [1-3].
Parenchymal thickness gives an additional indication of the chronicity of renal failure beyond the renal length and it is regarded as a more exact sonographic parameter in end-stage renal failure as renal size [4,
5].
Although the change in RCT has been comprehensively studied in acut and chronic renal diseases,
there is a little knowledge in healthy people whether the normal RCT,
an index for studying the health status of the kidney,
is only influenced by the environment or genetic factors can also contribute to that trait.
We hypothetized that the well-known normal age-related change in RCT can be also genetically influenced [6].
Our purpose was to estimate heritability and environmental effects on the development of RPT.